ENERGETIC METABOLISM AND FATIGABILITY IN EXPERIMENTAL MYOTONIA

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dc.contributor.authorRAMIREZ, BU
dc.date.accessioned2025-01-23T19:23:14Z
dc.date.available2025-01-23T19:23:14Z
dc.date.issued1990
dc.description.abstractExperimental myotonia was induced in rats by 2,4-dichloro-phenoxyacetic acid (2,4-D). After 4 to 24 h of treatment, the anterior tibialis muscles exhibited increased fatigue at low frequency (30 Hz) nerve stimulation, but they developed normal tension at high-frequency (100 Hz) stimulation. Glycogen content and the activities of glycogen phosphorylase, lactate dehydrogenase and malate dehydrogenase remained normal. The absence of correlation between fatigability and energetic metabolism in this experimental model of myotonia suggests a dysfunction in excitation-contraction coupling.
dc.fuente.origenWOS
dc.identifier.issn0004-0533
dc.identifier.urihttps://repositorio.uc.cl/handle/11534/99180
dc.identifier.wosidWOS:A1990DP03400003
dc.issue.numero1
dc.language.isoen
dc.pagina.final20
dc.pagina.inicio17
dc.revistaArchivos de biologia y medicina experimentales
dc.rightsacceso restringido
dc.subject.ods03 Good Health and Well-being
dc.subject.odspa03 Salud y bienestar
dc.titleENERGETIC METABOLISM AND FATIGABILITY IN EXPERIMENTAL MYOTONIA
dc.typeartículo
dc.volumen23
sipa.indexWOS
sipa.trazabilidadWOS;2025-01-12
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