D-lactoacidosis como Complicación del Síndrome de Intestino Corto

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dc.catalogadoraba
dc.contributor.authorArancibia Assael, Gabriel
dc.contributor.authorHodgson Bunster, María Isabel
dc.contributor.authorHarris D., Paul R.
dc.date.accessioned2024-01-17T19:36:08Z
dc.date.available2024-01-17T19:36:08Z
dc.date.issued2010
dc.description.abstractShort bowel syndrome is defined as the loss, congenital or acquired, anatomical or functional, of a large part of the small intestine that generates inadequate absorption of nutrients and the frequent need of prolonged parenteral nutrition. The etiology of short bowel is diverse and varies with age. The necrotizing enterocolitisis and the midgut volvulus are among the most frequent causes. The bacterial overgrowth is frequently observed in children with short bowel, due to the secondary dilation of the remaining small bowel and to the associated intestinal dysmotility. It is more frequent in absence of the ileocecal valve. We present a 6 year old boy with short bowel syndrome secondary to extensive intestinal resection after a volvulous of the medium small intestine, 9 months before admission to the hospital, and who was on cyclical parenteral nutrition at home. The child developed ataxia, disarthria, dizziness and conscience compromise been admitted to de intensive care unit in deep sopor. An extensive work up including metabolic, infectious, toxicology and SNC imaging was negative except for metabolic acidosis. He was discharged on good conditions. Even though the child was on supportive therapy, the patient was readmitted few hours later with similar symptoms. D-lactoacidosis was suspected and confirmed with a value of 6.69 mmol/l (normal range: 0,0-0,25). Literature about this uncommon complication and its mechanism is reviewed. D-lactoacidosis should be suspected in every patient with short bowel syndrome and unexplained metabolic acidosis associated with neurologic symptoms.
dc.fechaingreso.objetodigital2024-01-19
dc.format.extent5 páginas
dc.fuente.origenORCID-ene24
dc.identifier.doi10.4067/S0370-41062010000400010
dc.identifier.eissn0717-6228
dc.identifier.issn0370-4106
dc.identifier.scopusidSCOPUS_ID:78649811876
dc.identifier.urihttps://doi.org/10.4067/S0370-41062010000400010
dc.identifier.urihttps://repositorio.uc.cl/handle/11534/80585
dc.information.autorucEscuela de Medicina; Arancibia Assael, Gabriel; S/I; 1007339
dc.information.autorucEscuela de Medicina; Hodgson Bunster, María Isabel; S/I; 99461
dc.information.autorucEscuela de Medicina; Harris D., Paul R.; 0000-0001-6226-0957; 80706
dc.issue.numero4
dc.language.isoes
dc.nota.accesoContenido completo
dc.pagina.final357
dc.pagina.inicio353
dc.revistaRevista Chilena de Pediatría
dc.rightsacceso abierto
dc.rights.licenseCC BY 4.0 DEED Attribution 4.0 International
dc.rights.urihttps://creativecommons.org/licenses/by/4.0/
dc.subjectD-lactic acidosis
dc.subjectParenteral nutrition
dc.subjectShort bowel
dc.subject.ddc610
dc.subject.deweyMedicina y saludes_ES
dc.titleD-lactoacidosis como Complicación del Síndrome de Intestino Corto
dc.title.alternativeD-lactoacidosis as short bowel syndrome complication
dc.typeartículo
dc.volumen81
sipa.codpersvinculados1007339
sipa.codpersvinculados99461
sipa.codpersvinculados80706
sipa.indexWoS
sipa.indexPubmed
sipa.indexSCOPUS
sipa.indexScielo
sipa.indexmedẽs
sipa.indexLatindex
sipa.trazabilidadORCID;2024-01-08
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