Browsing by Author "KASE, CS"

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    CYSTICERCOSIS OF THE CENTRAL NERVOUS-SYSTEM - CLINICAL AND THERAPEUTIC CONSIDERATIONS
    (1984) TORREALBA, G; DELVILLAR, S; TAGLE, P; ARRIAGADA, P; KASE, CS
    In a group of 40 cases of cysticercosis of the CNS, 59% presented with intracranial hypertension due to obstructive hydrocephalus. Ventricular or cisternal cysts, and chronic cysticercus meningitis were the most common causes of hydrocephalus. Seizures occurred in 40% of the patients, in one-half of them in association with CT-detected parenchymatous cysts. In 20% of the cases progressive mental deterioration was the main clinical feature, at times associated with hydrocephalus. CT scan provided the highest diagnostic yield, being abnormal in 90% of cases. Long term prognosis was poor, with a mortality rate of 38% over a 40-mo. follow-up period. The most common cause of death (60%) was meningitis. CSF shunting is the treatment of choice for hydrocephalus, irrespective of its mechanism. Surgical resection is indicated in some cases with a single superficial (cortical) or posterior fossa cyst. Supratentorial cysts carry a relatively benign prognosis.
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    GLOBAL SPATIAL DISORIENTATION - CLINICOPATHOLOGIC CORRELATIONS
    (1977) KASE, CS; TRONCOSO, JF; COURT, JE; TAPIA, JF; MOHR, JP
    Patients (2) presenting with the acute onset of bilateral parietal lobe damage showed initially the features of Balint''s syndrome. After most of its manifestations had cleared, both patients exhibited severe disorders of spatial orientation: acoustic ataxia and inability to localize objects in space. One patient showed a topographical disorientation and abnormalities of whole body movements. This patient came to post-mortem examination, which revealed bilateral and fairly symmetrical old and recent infarctions of the superior parietal lobules. The reported abnormalities of whole body movements may be explained on the basis of a visuo-motor intrahemispheric disconnection due to the bilateral lesion of the dorsal parietal lobe. The gloval spatial disorientation may be the manifestation of a derangement of a specific function centered in the parietal lobe. Topographic disorientation apparently does not require the concomitant failure of oculomotor mechanisms for its production. The topography of the anatomical lesion was considered in relation to current concepts on brain damage after severe hypotension and cardiac arrest. This group of patients is most likely at risk of developing behavioral abnormalities akin to those reported.
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